Anatomy & Physiology: The Cardiovascular System I - The Heart
Terms in this set (22)
The pericardium has two parts: the fibrous pericardium and the serous pericardium. The serous pericardium has two layers: parietal and visceral (epicardium), with pericardial fluid between them.
The heart is located in the mediastinum, anterior to the vertebral column, posterior to the sternum, and superior to the diaphragm, with about two-thirds of its mass to the left of the midline.
The right and left coronary arteries branch off the aorta and supply oxygen-rich blood to the heart muscle tissue.
The coronary sinus collects oxygen-poor blood from the cardiac veins and returns it to the right atrium.
The heart has four chambers: two atria (right and left) that receive blood, and two ventricles (right and left) that pump blood out.
AV valves (tricuspid on right, bicuspid/mitral on left) separate atria from ventricles and prevent backflow during ventricular contraction.
SL valves (pulmonary on right, aortic on left) separate ventricles from arteries and prevent backflow after blood is ejected.
Blood flows: body → right atrium → tricuspid valve → right ventricle → pulmonary semilunar valve → pulmonary trunk → lungs → left atrium → bicuspid valve → left ventricle → aortic semilunar valve → aorta → body.
The pulmonary circuit carries blood between the heart and lungs; the systemic circuit carries blood between the heart and the rest of the body.
Cardiac muscle is striated, involuntary, with branched cells containing one nucleus, abundant mitochondria, and intercalated discs with gap junctions and desmosomes.
About 1% of cardiac muscle cells are autorhythmic pacemaker cells that generate spontaneous action potentials due to Na+ leakiness.
Includes the sinoatrial (SA) node, atrioventricular (AV) node, AV bundle (Bundle of His), and Purkinje fibers, which coordinate heart rhythm.
P wave: atrial depolarization; QRS complex: atrial repolarization and ventricular depolarization; T wave: ventricular repolarization.
Bradycardia (slow HR), tachycardia (fast HR), AV block (slow conduction), atrial fibrillation (no P wave), ventricular fibrillation (no organized waves, fatal).
Atrial systole (atria contract), ventricular systole (ventricles contract), and relaxation (diastole) with corresponding valve openings and closings.
Lubb (1st sound) caused by closing of AV valves at ventricular systole start; Dupp (2nd sound) caused by closing of semilunar valves at ventricular systole end.
CO is the volume of blood pumped by a ventricle per minute. Formula: CO = HR × SV, where HR is heart rate and SV is stroke volume.
Increased preload (ventricular stretch) leads to increased force of contraction and stroke volume.
Preload (stretch), afterload (arterial pressure to overcome), and contractility influence stroke volume.
Sympathetic stimulation increases HR and contractility; parasympathetic (vagus nerve) decreases HR.
Hormones (epinephrine, norepinephrine, thyroid), electrolytes (Ca++, Na+, K+), body temperature, age, and fitness affect heart rate.
CHF is reduced cardiac output due to diseased heart muscle, causing pulmonary and peripheral edema; treatment includes activity, drugs, pacemakers, stents, or transplant.