Table of contents

1. Introduction to Biochemistry4h 34m
2. Water3h 23m
3. Amino Acids8h 10m
4. Protein Structure10h 4m
5. Protein Techniques14h 5m
6. Enzymes and Enzyme Kinetics13h 38m
7. Enzyme Inhibition and Regulation 8h 42m
8. Protein Function 9h 41m
9. Carbohydrates7h 49m
10. Lipids5h 49m
11. Biological Membranes and Transport 6h 37m
12. Biosignaling9h 45m
Review 1: Nucleic Acids, Lipids, & Membranes2h 47m
Review 2: Biosignaling, Glycolysis, Gluconeogenesis, & PP-Pathway3h 12m
Review 3: Pyruvate & Fatty Acid Oxidation, Citric Acid Cycle, & Glycogen Metabolism2h 26m
Review 4: Amino Acid Oxidation, Oxidative Phosphorylation, & Photophosphorylation1h 48m

10. Lipids

Fatty Acids

Biochemistry

10. Lipids

Fatty Acids

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Multiple Choice

Which of the following is NOT true concerning the actions of free fatty acids (FFAs)?

FFAs can be oxidized in mitochondria to produce ATP.

FFAs can serve as an important energy source during prolonged fasting.

Elevated levels of FFAs in the blood can contribute to insulin resistance.

FFAs are directly converted into glucose via gluconeogenesis.

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Verified step by step guidance

Step 1: Begin by understanding the role of free fatty acids (FFAs) in metabolism. FFAs are released from triglycerides during lipolysis and serve as an energy source, especially during fasting or prolonged exercise.

Step 2: Recall the process of fatty acid oxidation. FFAs are transported into mitochondria, where they undergo β-oxidation to produce acetyl-CoA. Acetyl-CoA enters the citric acid cycle, leading to ATP production. This confirms that FFAs can be oxidized in mitochondria to produce ATP.

Step 3: Consider the role of FFAs during prolonged fasting. During fasting, FFAs are mobilized from adipose tissue and used as an energy source by tissues such as muscle and liver. This supports the statement that FFAs serve as an important energy source during fasting.

Step 4: Examine the relationship between elevated FFAs and insulin resistance. High levels of FFAs in the blood can interfere with insulin signaling pathways, contributing to insulin resistance. This is a well-documented phenomenon in metabolic disorders.

Step 5: Address the incorrect statement. FFAs cannot be directly converted into glucose via gluconeogenesis. Gluconeogenesis primarily uses substrates like lactate, glycerol, and amino acids, but not FFAs. FFAs are metabolized into acetyl-CoA, which cannot serve as a precursor for glucose synthesis due to the irreversible nature of the pyruvate dehydrogenase reaction.