Biochemistry: Carbohydrate Metabolism and Glucose Pathways
Terms in this set (23)
Glucose is the major fuel for the body, converted to pyruvate and then acetyl-SCoA for ATP production.
Digestion begins in the mouth with salivary α-amylase and ends in the small intestine where disaccharides are hydrolyzed to monosaccharides.
α-Amylase catalyzes hydrolysis of α-glycosidic bonds, producing maltose and smaller polysaccharides.
Phosphorylation traps glucose inside the cell and commits it to metabolism; phosphorylated glucose cannot cross the membrane.
Glycolysis, glycogenesis (storage as glycogen), fatty acid synthesis, and entry into the pentose phosphate pathway.
Conversion of glucose to two pyruvates, production of two ATPs, and two NADH molecules.
Steps 1 and 3: phosphorylation of glucose to glucose-6-phosphate and fructose-6-phosphate to fructose-1,6-bisphosphate.
It catalyzes step 3, a major control point, activated by ADP/AMP and inhibited by ATP and citrate.
It is cleaved into two three-carbon sugars: glyceraldehyde 3-phosphate and dihydroxyacetone phosphate.
Pyruvate is reduced to lactate to regenerate NAD+, allowing glycolysis to continue.
Converts pyruvate to acetyl-SCoA, releasing CO2 and producing NADH, requiring multiple enzymes and cofactors.
Transfers electrons from cytosolic NADH to mitochondrial FAD, producing 2 ATP per NADH during oxidative phosphorylation.
It transfers electrons from cytosolic NADH to mitochondrial NADH, yielding 3 ATP per NADH, and involves malate and aspartate transport.
Insulin lowers blood glucose by promoting uptake and storage; glucagon raises glucose by stimulating glycogen breakdown and gluconeogenesis.
Glycogenolysis speeds up, gluconeogenesis increases, and ketone bodies are produced from acetyl-SCoA as fat metabolism rises.
Ketone bodies are produced from excess acetyl-SCoA and serve as alternative energy sources for brain and tissues.
Insufficient insulin or insulin resistance leads to high blood glucose, glucose in urine, and cellular glucose deprivation.
Type I is insulin-dependent due to pancreatic beta cell destruction; type II is insulin-resistant, often linked to obesity.
The synthesis of glycogen from glucose-6-phosphate via glucose-1-phosphate and UDP-glucose.
Glycogen phosphorylase releases glucose-1-phosphate from glycogen, which is converted to glucose-6-phosphate.
The synthesis of glucose from noncarbohydrate precursors like lactate, amino acids, and glycerol, mainly in the liver.
Because steps 1, 3, and 10 are highly exergonic and irreversible, gluconeogenesis uses alternative enzymes and reactions.
The recycling of lactate produced by muscles during anaerobic glycolysis back to glucose in the liver.