Table of contents

1. Introduction to Genetics51m
2. Mendel's Laws of Inheritance3h 37m
3. Extensions to Mendelian Inheritance2h 41m
4. Genetic Mapping and Linkage2h 28m
5. Genetics of Bacteria and Viruses1h 21m
6. Chromosomal Variation1h 48m
7. DNA and Chromosome Structure56m
8. DNA Replication1h 10m
9. Mitosis and Meiosis1h 34m
10. Transcription1h 0m
11. Translation58m
12. Gene Regulation in Prokaryotes1h 19m
13. Gene Regulation in Eukaryotes44m
14. Genetic Control of Development44m
15. Genomes and Genomics1h 50m
16. Transposable Elements47m
17. Mutation, Repair, and Recombination1h 6m
18. Molecular Genetic Tools19m
- Genetic Cloning
  9m
- Methods for Analyzing DNA
  9m
19. Cancer Genetics29m
- Overview of Cancer
  21m
- Cancer Mutations
  7m
20. Quantitative Genetics1h 26m
21. Population Genetics50m
- Hardy Weinberg
  19m
- Allelic Frequency Changes
  31m
22. Evolutionary Genetics29m

3. Extensions to Mendelian Inheritance

Overview of interacting Genes

Genetics

3. Extensions to Mendelian Inheritance

Overview of interacting Genes

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Multiple Choice

Polygenic traits are controlled through which of the following ways.

Multiple alleles for one gene

Multiple genes

Epistasis

Penetrance

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Verified step by step guidance

Understand the concept of polygenic traits: These are traits that are controlled by more than one gene, often resulting in a continuous range of phenotypes.

Recognize that polygenic traits are influenced by multiple genes, each contributing to the overall phenotype. This is different from traits controlled by a single gene with multiple alleles.

Consider examples of polygenic traits, such as human height or skin color, which are influenced by the combined effect of several genes.

Differentiate polygenic inheritance from epistasis, where one gene can affect or mask the expression of another gene, and penetrance, which refers to the proportion of individuals carrying a particular variant of a gene that also express an associated trait.

Conclude that polygenic traits are controlled by multiple genes, which collectively influence the phenotype, rather than by multiple alleles of a single gene or through epistasis or penetrance.