Table of contents

1. Introduction to Anatomy & Physiology5h 43m
2. Cell Chemistry & Cell Components12h 36m
3. Energy & Cell Processes10h 6m
4. Tissues & Histology10h 3m
5. Integumentary System2h 20m
6. Bones & Skeletal Tissue2h 16m
7. The Skeletal System2h 35m
8. Joints2h 17m
9. Muscle Tissue2h 33m
10. Muscles1h 11m
11. Nervous Tissue and Nervous System1h 35m
12. The Central Nervous System1h 6m
- Introduction to the Central Nervous System
  18m
- The Cerebrum
  48m
13. The Peripheral Nervous System1h 26m
14. The Autonomic Nervous System1h 38m
15. The Special Senses2h 41m
16. The Endocrine System2h 48m
17. The Blood3h 22m
18. The Heart3h 42m
19. The Blood Vessels3h 35m
20. The Lymphatic System3h 16m
21. The Immune System14h 37m
22. The Respiratory System3h 20m
23. The Digestive System2h 5m
24. Metabolism and Nutrition4h 0m
25. The Urinary System2h 39m
26. Fluid and Electrolyte Balance, Acid Base Balance37m
27. The Reproductive System2h 5m
28. Human Development1h 21m
29. Heredity3h 32m

25. The Urinary System

Introduction to the Urinary System

Anatomy & Physiology

25. The Urinary System

Introduction to the Urinary System

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Multiple Choice

What is the primary pathophysiologic basis of acute glomerulonephritis?

Degeneration of renal tubular cells

Obstruction of the ureters by kidney stones

Excessive secretion of antidiuretic hormone

Immune-mediated inflammation of the glomeruli

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Verified step by step guidance

Understand the term 'acute glomerulonephritis': Acute glomerulonephritis is a condition characterized by inflammation of the glomeruli, which are the tiny filtering units in the kidneys responsible for removing waste and excess fluid from the blood.

Recognize the pathophysiologic basis: The primary cause of acute glomerulonephritis is immune-mediated inflammation. This occurs when the immune system mistakenly targets the glomeruli, leading to damage and impaired kidney function.

Identify the mechanism of immune-mediated inflammation: Antigen-antibody complexes are deposited in the glomeruli, triggering an inflammatory response. This response involves the activation of immune cells and the release of inflammatory mediators, which damage the glomerular structure.

Differentiate from other options: Degeneration of renal tubular cells, obstruction of the ureters by kidney stones, and excessive secretion of antidiuretic hormone are unrelated to the immune-mediated inflammation seen in acute glomerulonephritis. These options describe other pathophysiologic processes.

Conclude the correct answer: Based on the understanding of the immune-mediated mechanism, the correct answer is 'Immune-mediated inflammation of the glomeruli,' which is the primary pathophysiologic basis of acute glomerulonephritis.